A synthetic glucocorticoid with little mineralocorticoid action. It has potent anti-inflammatory action and alters body immune responses. Given systemically triamcinolone exhibits the typical glucocorticoid acitvity including negative feedback suppression of the pituitary-adrenal axis. This may result in corticosteroid deficency in stress situations.
Bronchial asthma, allergies, dermatoses, rheumotoid, arthritis and other musuclo-skeletal inflamatory conditions, connective tissue disorders, and inflammatory joint disease.
Local/systemic fungal infections, septic arthritis herpes simplex, hypersens, lactation, peptic ulcer. Osteoporosis (long term use), H/o glucocorticoid induced myopathy.
Acute adrenal insufficiency may be precipitated by infection/trauma in patients on long term corticosteroid therapy. Peptic ulceration, haemorrhage and perforation, glaucoma, hyperglycaemia, G.I.upsets, nocturia, increased appetite, obsesity, facial rounding, increased fragility of skin, behavioural changes.
Infection near inj. site, diabetes, hypertension, CVS disease, renal impairment, glaucoma, elderly psychosis, delayed tissue healing, neonates, children, pregnancy, liver cirrhosis.
10mg/ml
and 40mg/ml, vial.
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